Literaturverzeichnis

[1] Reiser KM, Last JA. Silicosis and fibrogenisis: fact and artifact. Toxicol. 1979; 13: 51-72.

[2] MSD Sharp&Dohme GmbH, Haar. MSD Manual . 6. Auflage, München, Jena: Urban & Fischer, 2000: 755ff.

[3] Könn G, Schejbal V, Oellig W-P. Pneumokoniosen In: Doerr W, Seifert G. (Hrsg.). Spezielle pathologische Anatomie – Pathologie der Lungen. Band 16/II. Berlin, Heidelberg, New York, Tokyo: Springer, 1983: 647-808.

[4] Gedigk P, Totovic V. Zell- und Gewebsschäden. In: Eder M, Gedigk P (Hrsg.). Allgemeine Pathologie und Pathologische Anatomie . 33. Auflage, Berlin, Heidelberg, New York: Springer, 1990: 6-74.

[5] Reichel G. Stäube. In: Reichel G, Bolt HM, Hettinger T et al. (Hrsg.). Grundlagen der Arbeitsmedizin. Stuttgart, Berlin, Köln, Mainz: W. Kohlhammer, 1985: 362-382.

[6] Südbeck C. Berufskrankheiten. In: Südbeck C. (Hrsg.). Arbeits- und Betriebsmedizin. 2. Auflage, Stuttgart, New York: Schattauer, 1998: 29-98.

[7] Reichel G. Auf anorganische Stäube mit geringem oder fehlendem Quarzgehalt zurückgehende Lungenveränderungen. In: Schwiegk H (Hrsg.). Handbuch der Inneren Medizin . Band 4. Berlin, Heidelberg, New York: Springer, 1976: 467-469.

[8] Lee KP, Trochimowicz HJ, Reinhardt CF. Pulmonary response of rats exposed to titanium dioxide (TiO2) by inhalation for two years . Toxicol Appl Pharmacol. 1985; 79: 179-192.

[9] Riede UN, Costabel U. Lunge. In: Riede UN , Schaefer HE, Wehner H (Hrsg.). Allgemeine und Spezielle Pathologie . 2. Auflage, Stuttgart, New York: Georg Thieme, 1989: 554-591.

[10] Banks DE. Clinical features of silicosis. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 23-37.

[11] Seidel HJ. Arbeitsmedizin. In: Gaus W, Hingst V, Mattern R, Reinhardt G, Seidel HJ, Sonntag H-G (Hrsg.). Ökologisches Stoffgebiet . 3. Auflage, Stuttgart: Hippokrates bei Georg Thieme, 1999: 194.

[12] Nash T, Allison AC, Harington JS. Physico-chemical properties of silica in relation to its toxicity. Nature. 1966; 210: 259-261.

[13] Kane AB. Questions and controversies about the pathogenesis of silicosis. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 121-136.

[14] Allison AC. Fibrogenic and other biological effects of silica. Curr Top Microbiol Immunol. 1996; 210: 147-158.

[15] Castranova V, Dalal NS, Vallyathan V. Role of Surface free radicals in the pathogeicity of silica. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 91-105.

[16] Fubini B et al .. Physico- chemical properties of crystalline silica dusts and their implication in various biological responsese. Scan J Work Environ Health 1995; 21(2): 9-14.

[17] Stalder K, Stöber W. Hemolytic activity of suspension of different silica modification and inert dust. Nature. 1965; 206: 874-875.

[18] Allison AC. Lysosomes and the toxicity of particulate pollutants. Arch Intern Med. 1971; 128: 131-139.

[19] Nadler S, Goldfischer S. The intracellular release of lysosomal contents in macrophages that have ingested silica. J Histochem Cytochem. 1970; 18: 368.

[20] Jabbour AJ et al .. Potential intracellular messengers involved in silica stimulation of alveolar macrophages. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 137-149.

[21] Schmidt JA et al .. Silica- stimulated monocytes release fibroblast proliferation factors identical to interleukin 1. J Clin Invest. 1984; 73: 1462.

[22] Green FHY, Vallyathan V. Pathologic responses to inhaled silica. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 39-59.

[23] Kane AB. Particle- and fiber- induced lesions: an overview. In: Mohr U, Dungworth DL, Mauderly JL, Oberdörster G (Hrsg.). Toxic and carcinogenic effects of solid particles in the respiratory tract. Washington, D.C.: ILSI Press, 1994: 3-16.

[24] Fogekmark B et al. Pulmonary macrophage phagocytosis and enzyme production after in vivo exposure to silica dust. Toxicol Appl Pharmacol. 1983; 68: 152.

[25] Fischer GL, Wilson FD. The effects of coal fly ash and silica inhalation of macrophage function and progenitors. J Reticuloendoth Soc. 1980;

[26] Keane MJ, Wallace WE. Pulmonary surfactant adsorption and the expression of silica toxicity. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 271- 281.

[27] Wallace WE et al .. Contrasting respirable quartz and kaolin retention of lecithin surfactant and expression of membranolytic activity following phospholipase A2 digestion . J Toxicol Environ Health. 1992; 37: 391.

[28] Schlipköter HW, Brockhaus A. Die Hemmung der experimentellen Silikose durch subkutane Verabreichung von PVPNO. Klin Wochenschr. 1961; 39: 1182- 1189.

[29] Castranova V. Suppression of the cytotoxicity and fibrogenicity of silica with PVPNO. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 293-298.

[30] Nolan RP et al .. Quartz hemolysis as related to its surface functionalities. Environ Res. 1981; 210: 259-261.

[31] Allison AC, Harrington JS, Birbeck M. An examination of the cytotoxic effects of silica on macrophages. J Exp Med. 1966; 124: 141-154.

[32] Aufenanger J, Kattermann R. Lipid- und Lipoproteinstoffwechsel. In: Greiling H, Gressner AM (Hrsg.). Lehrbuch der Klinischen Chemie und Pathobiochemie. 3.Auflage, Stuttgart, New York: Schattauer, 1995: 300-360.

[33] Stryer L. Die Biosynthese von Membranlipiden und Steroidhormonen. In: Stryer L. Biochemie. 4.Auflage, Heidelberg, Berlin, Oxford: Spektrum, 1996: 719-825.

[34] Linnemann M, Kühl M. Transport von Lipiden: Lipoproteine. In: Linnemann M, Kühl M. Biochemie für Mediziner. 4. Auflage, Braunschweig, Wiesbaden: Vieweg, 1995: 338-343.

[35] Brown MS, Goldstein JL. Ein rezeptor- vermittelter Stoffwechselweg für die Cholesterin- Homöostase (Nobel- Vortrag). Angew Chem. 1986; 7: 579-658.

[36] Schaefer H-E. Ätiologie und Pathogenese arterieller Verschlußkrankheiten. In: Alexander K (Hrsg.). Gefäßkrankheiten. 1994: 25-46.

[37] Glomset JA. The plasma lecithins: cholesterol acyltransferase reaction. J Lipid Res. 1968; 9: 155-167.

[38] Johnson WJ et al.. Cholesterol transport between cells and high-density lipoproteins. Biochem Biophys Acta. 1991; 1085: 273-298.

[39] Rothblat GH et al .. Apolipoproteins, membrane cholesterol domains, and the regulation of cholesterol efflux. J Lipid Res. 1992; 33: 1091-1097.

[40] Roosbeek S et al .. Three arginine residues in apolipoprotein A-I are critical for activation of lecithin :cholesterol acyltransferase. J Lipid Res. 2001; 42: 31-40.

[41] Rothblat GH et al. Cell cholesterol efflux: integration of old and new observations provides new insights. J Lipid Res. 1999; 40: 781-796.

[42] Oram JF, Yokoyama S. Apolipoprotein-mediated removal of cellular cholesterol and phospholipids. J Lipid Res. 1996; 37: 2473-2491.

[43] Löffler G. Lipide. In: Löffler G (Hrsg.). Basiswissen Biochemie mit Pathobiochemie. 3. Auflage, Berlin, Heidelberg, New York: Springer, 1999: 149-217.

[44] Acton S et al .. Identification of scavenger receptor SR-BI as a high density lipoprotein receptor. Science. 1996; 271: 518-520.

[45] Calvo D, Vega MA. Identification, primary structure, and distribution of CLA-1, a novel member of the CD36/LIMPII gene family. J Biol Chem. 1993; 268: 18929-18935.

[46] Mingpeng S, Zongli W. The protective role of high- density lipoproteins in artherosklerosis. Exp Gerontol. 1999; 34: 539-548.

[47] Rigotti A, Krieger M. Getting a handle on “good” cholesterol with the high- density lipoprotein receptor. N Engl J Med. 1999; 341: 2011-2013.

[48] Kannel BW et al .. Serum cholesterol, lipoproteins, and the risk of coronary heart disease. the framingham study. Ann Intern Med. 1971; 74: 1-12.

[49] Gordon DJ, Rifkind BM. High density lipoprotein. The clinical implications of recent studies. N Engl J Med. 1989; 321: 1311-1316.

[50] Parthasarathy S, Barnett J, Fong LG. High- density lipoprotein inhibits the oxidative modification of low- density lipoprotein. Biochem Biophys Acta. 1990; 1044: 275-283.

[51] Mertens A, Holvoet P. Oxidized LDL and HDL: antagonists in atherothrombosis. Faseb J. 2001; 15: 2073-2084.

[52] Quarck R et al .. Adenovirus- mediated gene transfer of human platelet- activating factor- acetylhydrolase prevents injury-induced neointima formation and reduces spontaneous atherosclerosis in apolipoprotein E- deficient mice. Circulation 2001; 103: 2495-2500.

[53] Laplaud PM, Dantoine T, Chapman MJ. Paraoxonase as a risk marker for cardiovascular disease: facts and hypotheses. Clin Chem Lab Med 1998; 36(7): 431-441.

[54] Navab M et al. Normal high density lipoprotein inhibits three steps in the formation of mildly oxidized low density lipoprotein: step 1. J Lipid Res. 2000; 41: 1481-1494.

[55] Navab M et al. Normal high density lipoprotein inhibits three steps in the formation of mildly oxidized low density lipoprotein: steps 2 and 3. J Lipid Res. 2000; 41: 1495-1508.

[56] Vohl MC et al. A novel lecithin- cholesterol acyltransferase antioxidant activity prevents the formation of oxidized lipids during lipoprotein oxidation. Biochemistry 1999; 38: 5976-5981.

[57] Karpe F. Postprandial lipoprotein metabolism and artherosclerosis. J Intern Med. 1999; 246: 341-355.

[58] Rüttner JR, Isler KM. Die Adsorption von Serumeiweißkörpern an Quarz und andere Mineralien. Schweiz med Wochenschr. 1956; 86: 63-67.

[59] McFee DR, Tye R. Adsorption of proteins on dust related to pneumoconiosis: selectivity. J occup Med. 1965; 7: 269-275.

[60] Noishiki Y. Application of immunoperoxidase method to microscopic observation of plasma protein on polymer surface. J Biomed Mater Res. 1982; 16: 359-367.

[61] Breemhaar W et al .. Preferential adsorption of HDL from blood onto biomaterial surface. Biomaterials . 1984; 5: 269-274.

[62] Pott F. Die krebserzeugende Wirkung anorganischer Fasern im Tierexperiment – Daten und Bewertung. Umwelthygiene. 1987/88; 20: 97-133.

[63] Aalto M, Hepplesto AG. Fibrogenesis by mineral fibres: an in-vitro study of the roles of the macrophage and fibre length. Br J exp Path. 1984; 65: 91-99.

[64] Chapman J. Comparative analysis of mammalian plasma lipoproteins. Methods Enzymol. 1986; 128: 70-143.

[65] Alexander C, Day CE. Distribution of serum lipoproteins of selected vertebrates . Comp Biochem Physiol. 1973; 46b: 295-312.

[66] Reichel G. Pneumokoniosen durch anorganische Stäube. In: Ferlinz R (Hrsg.). Pneumonologie in Praxis und Klinik. Stuttgart, New York: Georg Thieme, 1994: 581-608.

[67] Saffiotti U et al .. Carcinogenesis by cristalline silica: Animal, cellular and molecular studies. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 345- 381.

[68] Rüttner JR, Bovet P, Aufdermaur M. Graphit, Carborund, Staublunge. Deut Med Wochenschr. 1952; 77: 1413-1415.

[69] Konitzko H et al.. Lungenfibrosen bei der Bearbeitung von Hartmetallen. Deut Med Wochenschr. 1980; 105: 120-123.

[70] Rosmanith J et al .. The fibrogenic action of kaolinite, muscovite and feldspat. Silicosis Report/ North Rhine Westphalia. 1989; 17: 305-322.

[71] Sheers G. Prevalence of pneumoconiosis in Cornish kaolin workers. Brit J. Industr Med. 1964; 21: 218-225.

[72] Vallayathan V, Craighead JE. Pulmonary pathology in workers exposed to nonasbestiform talc. Human Pathol. 1984; 12: 28-35.

[73] Davis WB, Crystal RG. Cronic interstitial lung disease. In: Simmons DH (Hrsg.). Current pulmonology. Band 5 , Chicago : III, Year- Book Med. Publ., 1984 : 347-473.

[74] Wagner JC et al .. Animal experiments with talc. In: Inhaled particles. Band 4. Oxford: Pergammon Press, 1975: 647-654.

[75] Pickrell JA et al .. Talc deposition and effects after 20 days of repeated inhalation exposure of rats and mice to talc. Environ Res. 1989; 49: 233-245.

[76] Lundgren KD, Swensson A. Experimental investigations using the method of Miller and Sayers on the effect upon animals of cemented tungstencarbides, and the powders used as raw material. Acta Med Scand. 1953; 145: 20-27.

[77] Stalder K. Pathogenese und gewerbshygienische Aspekte der Silikose. Klin Wochenschr. 1968; 46: 457-464.

[78] Driscoll KE. The role of interleucin- I and tumor necrosis factor α in the lung`s response to silica. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 163-184.

[79] Nolan RP, Langer AM, Weisman I et al.. Surface character and membranolytic activity of rutile and anatase: two titanium dioxide polymorphs. Brit J Industr Med. 1987; 44: 687-698.

[80] Pott F, Friedrichs KH. Tumoren der Ratte nach i.p- Injektion faserförmiger Stäube. Naturwiss. 1972, 59: 318.

[81] Stacy BD et al .. Tissue changes in rats lungs caused by hydroxides, oxides and phosphates of aluminium and iron. J Pathol Bact . 1959; 7: 417-428.

[82] Nagelschmidt G et al .. The recovery of quartz and other minerals from the lungs of rats. Arch Industr Health. 1957; 16: 188-202.

[83] Brunauer S, Emmett PH, Teller E. Adsorption of gasaes in multimolecular layers. J Amer Chem Soc. 1938; 60: 309.

[84] Sing KSW. Adsorption methods for surface area determination. In: Stanley- Wood NG, Lines RW (Hrsg.). Particle size analysis. Cambridge: The Royal Society of Chemistry, 1992: 13-32.

[85] Bigelow WC, Pickett DL, Zisman WA. Oleophobic monolayers- films adsorbed from solution in non-polar liquids. J Colloid Sci. 1946; 1: 513-583.

[86] Kuhn H. Energieübertragung in monomolekularen Schichten. Naturwiss. 1967; 15/16: 429- 436.

[87] Davis WW, Krahl ME, Clowes GHA. Interactions between polycyclic hydrocarbons and sterols in mixed surface films at the air- water surface. J Am Chem Soc. 1940; 62: 3080-3098.

[88] Adam NK. The properties and molecular stucture of thin films of palmitic acid on water. Proc Roy Soc. 1921; 99(A): 336-351.

[89] Small DM. Lateral chain packing in lipids and membranes. J Lipid Res. 1984; 25: 1490-1500.

[90] Zocher H, Stiebel F. Dunkelfeldmikroskopie dünnster Filme auf Flüssigkeitsoberflächen. Z phys Chem Abt A. 1930; 147: 401- 435.

[91] Elshourbagy NA et al. The nucleotide and derived amino acid sequence of human apolipoprotein A-IV mRNA and the close linkage of its gene to the genes of apolipoproteins A-I and C-III. J Biol Chem. 1986; 261: 1998-2002.

[92] Daniel L et al .. Binding of the cationic dye, Janus Green B, as a measure of the specific surface area of crystalline silica in aqueous suspension. Toxicol Appl Pharmacol. 1993; 123: 62-67.

[93] Poot A et al .. Detection of surface- adsorbed (lipo )proteins by means of a two- step enzyme- immunoassay: A study on the Vroman effect. J Biomed Mater Res. 1990; 24: 1021-1036.

[94] Woodworth CD, Mossman BT, Craighead JE. Comparative effects of fibrous and nonfibrous minerals on cells and liposomes. Environ Res. 1982; 27: 190-205.

[95] Mahley RW et al .. Plasma lipoproteins: apolipoprotein structure and function. J Lipid Res. 1984; 25: 1277-1294.

[96] Frank PG, Marcel YL. Apolipoprotein A- I: structure- function relationship. J Lipid Res. 2000; 41: 853-872.

[97] Segrest JP et al.. The amphipathic helix in the exchangeable apolipoproteins: a review of secondary structure and function. J Lipid Res. 1992; 33: 141-166.

[98] Klimov AN et al .. On the mechanism of cholesterol interaction with apolipoproteins A-I and E. Chem Phys Lipids. 1992; 62: 229-237.

[99] Dickerson RE, Geis I. Struktur und Funktion der Proteine. Weinheim/Bergstraße: Chemie, 1971: 32.

[100] Strunz H. Mineralogische Tabellen. 4. Auflage, Leipzig: Geesr & Portig K.-G., 1966: 178,381,400.

[101] Assmann G. Lipidstoffwechsel und Arteriosklerose. Stuttgart, New York: Schattauer, 1982: 56.

[102] Rigotti A et al .. Scavenger receptor BI a cell surface receptor for high density lipoprotein. Curr Opin Immunol. 1997; 8: 181-188.

[103] La Ville AE et al .. In vitro oxidised HDL is recognised by the scavenger receptor of macrophages: implications for ist protective role in vivo. Atherosclerosis. 1994; 105: 179-189.

[104] Brown GM, Donaldson K. Modulation of quartz toxicity by aluminium. In: Castranova V, Vallyathan V, Wallace WE (Hrsg.). Silica and silica- induced lung diseases. Boca Raton, New York, London, Tokyo: CRC Press, Inc., 1996: 299-304.

[105] King EJ et al .. The action of different forms of pure silica on the lungs of rats. Brit J industr Med. 1953; 10: 9-17.

[106] Allison AC. Effects of silica, asbestos and other pollutants on macrophages. In: Brain JD, Proctor DF, Reid LM (Hrsg.). Air pollution and the lung. Jerusalem: Israel Universities Press, 1975: 114-132.

[107] Feng M et al .. Adsorption of high density lipoproteins (HDL) on solid surfaces. J Colloid Interface Sci. 1996; 177: 364-371.

[108] Van Damme HS et al .. Protein adsorption from plasma onto poly( n-alkylmethacrylate) surfaces. J Biomater Sci Polym Edn. 1991; 3: 69-84.

[109] Behling CA, Spector M. Quantitative characterization of cells at the interface of long- term implants of selected polymers. J. Biomed Mater Res. 1986; 20: 653-666.


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