[Seite 21↓]

Literaturangaben zu 3.2.

Allen, D.G., Orchard, C.H. Myocardial contractile function during ischemia and hypoxia. Circ. Res. 60:153-168,1987.

Anderson, P.G., Allard, M.F., Thomas, G.D., Bishop, S.P. Digerness, S.B. Increased ischemic injury but decreased hypoxic injury in hypertrophied rat hearts. Circ. Res. 67:948-959,1990.

Cooley, D.A., Reul, G.J., Wukasch, D.A. Ischemic contracture of the heart: „stone heart”. Am. J. Cardiol. 29:575-577,1972.

Do, E., Baudet, S., Gow, I.F., Ellis, D., Noireaud, J. Intracellular pH during hypoxia in normal and hypertrophied right ventricle of ferret heart. J. Mol. Cell. Cardiol. 27:927-939,1995.

Hutter, J.J., Mestril, R., Tam, E.K., Sievers, R.E., Dillmann, W.H., Wolfe, C.L. Overexpression of heat shock protein 72 in transgenic mice decreased infarct size in vivo. Circulation 94:1408-1411,1996.

Kammermeier, H. High energy phosphate of the myocardium: Concentration vs. free energy change. Basic Res. Cardiol. 82(Suppl.2):31-36, 1987.

Kentish, J.C. The effects of inorganic phosphate and creatine phosphate on force production in skinned muscles from rat ventricle. J. Physiol. 370:585-604,1986.

Kilgore, J.L., Musch, T.I., Ross, C.R. Regional distribution of HSP70 proteins after myocardial infarction. Basic Res. Cardiol. 91:283-288,1996.

Lammerich, A., Bohm, J., Schimke, I., Wagner, K.D., Storch, E., Günther, J. Effects of hypoxia, simulated ischemia and reoxygenation on the contractile function of human atrial trabeculae. Mol. Cell. Biochem. 160/161:143-151, 1996.

Laser, A. Neubauer, S., Tian, R. Hu, K., Gaudron, P., Ingwall, J.S., Ertl, G. Long-term beta-blocker treatment prevents chronic creatine kinase and lactate dehydrogenase system changes in rat hearts after myocardial infarction. J. Am. Coll. Cardiol. 27:487-493, 1996.

Marber, M.S., Mestril, R., Chi, S.H. Sayen, M.R., Yellon, D.M., Dillmann, W.H. Overexpression of the rat inducible 70-kD heat shock protein in a transgenic mouse increases the resistance of the heart to ischemic injury. J. Clin. Invest. 95:1446-1456, 1995.

Maulik, N., Watanabe, W., Engelman, D.T., Engelman, R.M., Das, D.K. Oxidative stress adaptation improves postischemic ventricular recovery. Mol. Cell. Miochem. 144:67-74, 1995a.


[Seite 22↓]

Maulik, N., Watanabe, W., Engelman, D.T., Engelman, R.M., Kagan, V.E., Kisin, E., Tyurin, V., Cordis, G.A., Das, D.K. Myocardial adaptation to ischemia by oxidative stress induced by endotoxin. Am. J. Physiol. 269:C907-C916,1995b.

Osaki, J. Haneda, T., Kashiwagi, Y., Oi, S., Fukuzawa, J., Sakai, H., Kikuchi, K. Pressure-induced expression of heat shock protein 70 mRNA in adult rat hearts is coupled both to protein kinase A-dependent and protein kinase C-dependent systems. J. Hypertens. 16:1193-1200,1998.

Pizurki, L., Polla, B.S. cAMP modulates stress protein synthesis in human monocytes-macrophages. J. Cell. Physiol. 161:169-177,1994.

Wagner, K.D., Essmann, V., Mydlak, K., Wirth, M., Gmehling, G., Bohlender, J., Stauss, H.M., Günther, J., Schimke, I., Scholz, H. Decreased susceptibility of cardiac function to hypoxia-reoxygenation in renin-angiotensinogen transgenic rats. Am. J. Physiol. Regulatory Integrative Comp. Physiol. 283:R153-R160, 2002.

Wagner, K.D., Geil, D., Schimke, I., Stauss, H.M., Lammerich, A., Theres, H., Pfitzer, G., Vetter, R., Günther, J. Decreased susceptibility of contractile function to hypoxia/reoxygenation in chronic infarcted rat hearts. J. Mol. Cell. Cardiol. 30:2341-2353,1998.

Wagner, K.D., Gmehling, G., Günther, J., Stauss, H.M., Mydlak, K., Theres, H., Scholz, H., Schimke, I. Contractile function of rat myocardium is less susceptible to hypoxia/reoxygenation after acute infarction. Mol. Cell. Biochem. 228:49-55,2001.

Wagner, K.D., Gmehling, G., Günther, J., Theres, H., Mydlak, K., Schimke, I., Scholz, H. Time-dependent changes of the susceptibility of cardiac contractile function to hypoxia-reoxygenation after myocardial infarction in rats. Mol. Cell. Biochem. 241:125-133,2002.

Yoshida, T., Maulik, N., Engelman R.M., Ho, Y.S., Rousou, J.A., Flack, J.E. III, Deaton, D., Das, D.K. Glutathion peroxidase knockout mice are susceptible to myocardial ischemia reperfusion injury. Circulation 96(Suppl. II):216-220, 1997.

Younes, A., Schneider, J.M., Bercovici, J., Swynhedauw, B. Redistribution of creatine kinase isoenzymes in chronically overloaded myocardium. Cardiovasc. Res. 19:15-19, 1994.

Yu, H., Yokoyama, M., Asano, G. Time corse of expression and localisation of heat shock protein 72 in the ischemic and reperfused rat heart. Jpn. Circ. J. 63:278-287,1999.


© Die inhaltliche Zusammenstellung und Aufmachung dieser Publikation sowie die elektronische Verarbeitung sind urheberrechtlich geschützt. Jede Verwertung, die nicht ausdrücklich vom Urheberrechtsgesetz zugelassen ist, bedarf der vorherigen Zustimmung. Das gilt insbesondere für die Vervielfältigung, die Bearbeitung und Einspeicherung und Verarbeitung in elektronische Systeme.
DiML DTD Version 3.0Zertifizierter Dokumentenserver
der Humboldt-Universität zu Berlin
HTML-Version erstellt am:
19.03.2004